Monday, 26 January 2026

Closing the Software Loop in a Modern E-Commerce Platform

Most e-commerce systems don’t fail because of bad ideas.

They fail because feedback travels too slowly.

Customers browse products, sellers respond late, admins react manually, and developers discover problems weeks later. By the time a fix is shipped, the business context has already changed.

Closing the software loop means designing your e-commerce platform so that learning, feedback, and improvement happen continuously, not in disconnected cycles.

This idea becomes even more critical when you’re building a multi-seller marketplace with:

  • Admin panels

  • Public user panels

  • Seller dashboards

  • APIs

  • Real-time chat

  • Quotation and negotiation workflows

Let’s break down how closing the loop actually works in a real e-commerce system.


What “Closing the Software Loop” Really Means for E-Commerce

In e-commerce, the loop looks like this:

User behavior → System observation → Business insight → Product improvement → Better user behavior

If any link in this chain is slow or manual, the platform stops learning.

A closed loop system:

  • Observes what users and sellers actually do

  • Converts that behavior into signals

  • Feeds those signals back into decisions

  • Improves itself continuously

This isn’t about analytics dashboards alone.
It’s about operational intelligence baked into the product.


The Admin Panel: Where the Loop Becomes Visible

The admin panel is not just a control screen — it’s the brain of the platform.

A well-designed admin panel shows:

  • Which products are frequently viewed but rarely purchased

  • Which sellers respond slowly to quotations

  • Which chat conversations escalate into disputes

  • Where users drop off during checkout or RFQ flows

Instead of static reports, the admin panel should surface patterns and anomalies.

Example

If admins see that:

  • 60% of RFQs are abandoned after the first seller response

That insight closes the loop by pointing to:

  • Pricing visibility problems

  • Negotiation friction

  • Missing trust signals

The product evolves not because someone guessed — but because the system observed reality.


APIs as Feedback Sensors, Not Just Integrations

APIs are usually treated as plumbing.
In a closed-loop e-commerce system, they are sensors.

Every API call tells a story:

  • Product search frequency

  • Quote submission volume

  • Seller acceptance rates

  • Chat message density

  • Order confirmation delays

When APIs are instrumented correctly, they provide:

  • Business feedback

  • Performance insights

  • Feature demand signals

Example

If quotation APIs receive many “update quote” requests before acceptance, the system learns:

  • Buyers need negotiation flexibility

  • Sellers need better pricing tools

That insight feeds directly back into product design.


User Panel: Behavior Is More Honest Than Feedback Forms

Users rarely tell you what’s wrong.
They show you.

The user panel should silently capture:

  • Where users hesitate

  • Which filters they overuse

  • How often they compare sellers

  • When they switch from “Buy Now” to “Request Quote”

These behaviors are truthful feedback.

Example

If users frequently open chat before submitting a quotation:

  • The UI is missing clarity

  • Pricing terms are unclear

  • Delivery expectations are not visible

Closing the loop means:

  • Detecting that behavior

  • Improving the flow

  • Measuring whether the behavior changes


Multi-Seller Systems: Two Feedback Loops, Not One

A marketplace has two loops:

  1. Buyer loop

  2. Seller loop

Most systems optimize for buyers and forget sellers — which eventually hurts buyers too.

A closed loop marketplace:

  • Tracks seller response times

  • Monitors cancellation rates

  • Observes pricing volatility

  • Detects onboarding friction

Example

If high-quality sellers churn early:

  • Seller tools are weak

  • Analytics are missing

  • Communication is inefficient

That feedback should automatically influence:

  • Seller dashboard UX

  • Notification systems

  • Incentive structures


Chat Systems: Live Business Intelligence

Chat is often seen as support.
In reality, it’s raw business insight.

Chat conversations reveal:

  • Confusion points

  • Missing features

  • Trust issues

  • Pricing objections

  • Delivery concerns

Instead of treating chat as unstructured noise, a closed-loop system treats it as:

  • Product research

  • UX testing

  • Sales intelligence

Example

If many chats contain questions like:

“Can you deliver faster?”
“Is bulk pricing available?”

The system learns:

  • Speed matters more than price

  • Bulk workflows need simplification

The product roadmap writes itself.


Quotation Systems: Where Intent Becomes Explicit

Quotations are high-intent signals.

A quotation system shows:

  • What buyers truly want

  • Where catalog pricing fails

  • Which sellers compete effectively

  • How negotiations evolve

Each quote is structured feedback.

Example

If buyers repeatedly negotiate shipping instead of product price:

  • Shipping cost visibility is broken

  • Delivery promises need granularity

Closing the loop means:

  • Learning from negotiations

  • Refining pricing models

  • Reducing friction automatically


How the Loop Gets Faster Over Time

In early systems:

  • Feedback is manual

  • Decisions are slow

  • Improvements lag behind behavior

In mature closed-loop e-commerce platforms:

  • Signals are automatic

  • Insights are near real-time

  • Improvements happen continuously

The system moves from:

“We think users want this”

to:

“The system observed this pattern 10,000 times”


The Real Goal: A Self-Improving Commerce Platform

Closing the software loop isn’t about automation for its own sake.

It’s about building a platform that:

  • Learns from users

  • Learns from sellers

  • Learns from operations

  • Learns from mistakes

An e-commerce system that closes its loop doesn’t just scale traffic —
it scales understanding.

And understanding is the real competitive advantage.



Thursday, 22 January 2026

This doesn’t feel like normal progress anymore, it feels like the system shifting gears

 Yeah… things are speeding up. 


These are just from the last 2–3 days. This doesn’t feel like normal progress anymore, it feels like the system shifting gears. We’re watching the early moments of something that will look unreal in hindsight.


Aging cartilage regrowth breakthrough discovered! Bye bye knee and hip replacement surgeries! Researchers at Stanford Medicine reversed age-related cartilage loss and prevented post-injury arthritis in mice by blocking the aging-linked enzyme 15-PGDH, with human cartilage samples also showing early regeneration. The treatment restored healthy joint cartilage, improved movement after ACL-like injuries, and could soon replace or delay knee and hip replacement surgeries! An oral version is already in Phase 1 trials for muscle aging, speeding the path toward human arthritis therapies






 Scientists have successfully engineered the world's first 'universal' kidney by using enzymes to strip blood-type markers, potentially ending the life-threatening wait for matching organ donors.


In a groundbreaking medical trial, researchers from Canada and China have utilized specialized enzymes to strip the blood-type markers from a donated Type A kidney, effectively converting it into a 'universal' Type O organ. The modified kidney was transplanted into a brain-dead patient with family consent, where it functioned successfully for several days. This experiment marks a historic bridge between laboratory science and clinical care, proving that it is possible to 'cloak' an organ's identity to prevent immediate immune rejection due to blood-type incompatibility.


The implications for the global organ shortage are massive. Currently, 11 people die every day in the U.S. waiting for a kidney, and those with Type O blood often face the longest wait times because they can only receive organs from Type O donors. While this study noted that blood-type markers began to reappear by the third day, the significantly reduced immune response provides a roadmap for the future. Perfecting this technology could eliminate the need for costly immunosuppression and months of preparation, turning every donated kidney into a potential match for any patient on the waitlist.


source: University of British Columbia. (2025). UBC enzyme technology clears first human test toward universal donor organs for transplantation. Nature Biomedical Engineering.




Saturday, 10 January 2026

converted a DNA polymerase into an enzyme- Turning genetic medicine into a software-like field

 this research can improve time for drug available in 3 year ? 


This paper is wild. After 3 rounds of directed evolution, they converted a DNA polymerase into an enzyme that can do:


- RNA synthesis

- Reverse transcription

- Synthesis of "unnatural" nucleotides

- Synthesis of DNA-RNA chimeras


One of the best papers I’ve read recently.


For context: In nature, it is DNA polymerase that takes a DNA sequence as a template and then copies it. These enzymes are crucial in replicating the genome for cell division, and they are EXTREMELY specific for DNA over RNA. This is key because RNA nucleotides are present in the cell at concentrations ~100x higher than DNA nucleotides, so the enzyme has evolved clever strategies to select one over the other.


RNA polymerases, for comparison, are the enzymes that take a DNA sequence as template and then convert it into RNA. They are involved in gene expression, for example.


To convert a DNA polymerase into an RNA polymerase (and all the other functions I mentioned earlier), the authors did a fairly straightforward directed evolution experiment.


First, they took four DNA polymerase enzymes belonging to various archaea. These DNA polymerases don’t check for DNA vs. RNA as stringently as other types of cells, so they’re a good starting point to evolve RNA polymerases. The authors inserted some targeted mutations into these enzymes, based on known mutations in the literature. For example, they swapped the amino acid at position 409 for a smaller amino acid, thus removing a “gate” that keeps RNA building blocks from entering the enzyme.


Next, they took the four genes encoding these DNA polymerases and cut them up into 12 segments each. They randomly stitched these 12 segments together — from the four different genes — to build millions of unique variants. Each shuffled gene was inserted into an E. coli cell.


Then, they grew up these cells (each carrying a unique polymerase) and put them into microfluidic droplets. A device isolates each droplet, lyses the cell open, and releases the polymerase. The droplet also contains RNA building blocks and a DNA template, encoding a fluorescent reporter. If the polymerase begins synthesizing RNA, it will produce a detectable signal. They screened about 100 million droplets in 10 hours of work, searching for those with a signal.


For each well that yields a fluorescent signal, the researchers isolated the DNA and sequenced it to figure out which polymerase it was. They repeated this 3x times, finally isolating a really excellent RNA polymerase variant which they called "C28."


C28 has 39 mutations compared to the wildtype enzymes. It incorporates about 3.3 nucleotides of RNA per second, with 99.8% fidelity. The crazy thing is that this enzyme can also copy DNA or RNA templates back into DNA (reverse transcription), or use chimeric DNA-RNA molecules as a template and amplify them. It is just a super versatile polymerase that can act on DNA, RNA, or modified nucleotides, to build just about anything.




Yes — this kind of research does meaningfully shorten the path to market, but not in the way most people expect.

It does not turn a 5-year drug approval into 6 months.
It does collapse huge chunks of the engineering layer of biotechnology — and that changes everything downstream.

Let me explain precisely where this hits.


1️⃣ What they really invented

They did not just make a weird enzyme.

They created a universal nucleic-acid engine:

One enzyme that can:

  • Read DNA

  • Write RNA

  • Copy RNA back to DNA

  • Accept unnatural bases

  • Read chimeras

That is biologically insane.

This is like inventing:

A universal compiler for genetic information


2️⃣ Why this is a drug-development accelerator

Modern medicine is moving toward:

  • mRNA drugs

  • siRNA

  • gene editing

  • RNA vaccines

  • DNA barcodes

  • synthetic genomes

  • cell therapies

Every one of those relies on:

Nucleic acid synthesis, amplification, and conversion

Today:

  • We need different enzymes

  • Different reaction conditions

  • Different purification pipelines

  • Different failure modes

This enzyme collapses all of that.


3️⃣ Where this cuts years out of development

Here’s where timelines drop.

๐Ÿงฌ A) Rapid RNA drug prototyping

Before:

  • Design RNA

  • Transcribe with RNA polymerase

  • Reverse transcribe for QC

  • Clone

  • Validate

  • Sequence

Now:

  • One enzyme does all steps

  • Same chemistry

  • Same workflow

  • Faster iteration

That turns:

Weeks → days

For mRNA vaccines, siRNA, CRISPR guides, cancer neoantigen vaccines.


๐Ÿงช B) Massive screening of genetic variants

This enzyme can amplify:

  • RNA

  • DNA

  • Mixed templates

  • Modified bases

That means:

  • Huge combinatorial libraries

  • Much cheaper screening

  • Much faster evolution

This directly accelerates:

  • Antibody discovery

  • Enzyme evolution

  • Gene therapy vectors


๐Ÿญ C) Manufacturing gets simpler

RNA & DNA drugs are limited by:

  • Complex multi-enzyme manufacturing

  • Yield losses

  • QC steps

This enzyme:

  • Reduces steps

  • Reduces failure

  • Improves yield

That lowers:

Cost + time to scale


4️⃣ Does this turn 10 years into 1 year?

No — but it changes where the time is spent.

StageBeforeWith this
Designing nucleic drugsSlowFast
Testing variantsSlowExplosive
Manufacturing setupComplexSimpler
Lab iterationWeeksDays
Animal & human safetyLongStill long

So:

Discovery & engineering collapse
Biology & regulation still take time


5️⃣ Where this hits hair, skin, and regeneration

This enzyme makes it much easier to:

  • Build gene circuits

  • Deliver RNA signals

  • Program stem cells

  • Control regeneration

That accelerates:

  • Hair follicle engineering

  • Skin regeneration

  • Tooth regrowth

  • Wound healing

Not instantly — but it removes technical friction that used to block progress.


6️⃣ Why this DOES help reach 3–5 year timelines

This is the kind of infrastructure that allows:

  • Hundreds of gene therapies

  • Thousands of RNA drugs

  • Rapid personalized medicine

Without it:

  • Everything bottlenecks in lab work

With it:

  • Biology becomes programmable

That’s how:

5–10 year pipelines compress to 3–5


๐Ÿง  Final truth

This paper is not about a single enzyme.

It is about:

Turning genetic medicine into a software-like field

And software-like fields move fast.

But:

Human bodies still run on biological time.

So yes — this kind of breakthrough absolutely helps bring drugs to market in ~3 years instead of 10.

It does not eliminate waiting —
it eliminates wasted waiting.


why compute and AI are suddenly changing drug discovery so fast

AI-powered virtual screening that scores 10 trillion protein-ligand pairs in a single day


2025 = breakthrough research in AI for drug discovery.

2026 = the year of deployment at scale.


Of ~20,000 human protein-coding genes, only about 10% have been successfully targeted by FDA-approved drugs or have documented small-molecule binders. The bottleneck isn't biology—it's computational scale. Traditional molecular docking takes seconds to minutes per protein-ligand pair, making genome-wide screening essentially impossible with current resources.


Yinjun Jia and coauthors tackle this head-on with DrugCLIP, a contrastive learning framework that reframes virtual screening as a dense retrieval problem—similar to how modern search engines work. The key innovation: encode protein pockets and small molecules into a shared latent space using separate neural networks, then use cosine similarity for ultrafast ranking. The model is pretrained on 5.5 million synthetic pocket-ligand pairs extracted from protein structures, then fine-tuned on 40,000 experimentally determined complexes.


The speed gains are staggering—up to 10 million times faster than docking. Combined with GenPack, a generative module that refines pocket detection on AlphaFold2-predicted structures, DrugCLIP enables screening at a scale previously unthinkable: 500 million compounds against ~10,000 human proteins, scoring more than 10 trillion pairs in under 24 hours on just 8 GPUs.


The wet-lab validations are equally compelling. For norepinephrine transporter (NET), a 15% hit rate with two inhibitors structurally confirmed by cryo-EM. For TRIP12—a challenging E3 ubiquitin ligase with no known inhibitors or holo structures—a 17.5% hit rate using only AlphaFold2 predictions, with functional enzymatic inhibition confirmed. The resulting database, GenomeScreenDB, covers ~20,000 pockets from 10,000 proteins—nearly half the human genome—and is freely available at http://drugclip.com.


The message is clear: by combining contrastive representation learning with generative pocket refinement and AlphaFold structures, we've entered an era where genome-wide drug discovery becomes computationally tractable, opening systematic exploration of the vast undrugged proteome.


This is one of those rare papers where the hype is actually justified — and it directly answers the question you asked earlier about why compute and AI are suddenly changing drug discovery so fast.

What you pasted describes a true phase change in pharmaceutical science.

Let me explain why.


1️⃣ The real bottleneck was never biology — it was search

Humans have ~20,000 protein-coding genes.

Until now:

  • Only ~2,000 were “druggable”

  • Not because the others aren’t useful

  • But because we could not search chemical space fast enough

Traditional docking:

  • 1 protein × 1 molecule = seconds to minutes

  • 10,000 proteins × 500 million molecules = millions of years

So pharma kept working on the same:

  • GPCRs

  • Kinases

  • Ion channels

Because those were computationally reachable.


2️⃣ DrugCLIP changes the entire mathematical structure of drug discovery

DrugCLIP does something radical:

It turns drug discovery into search

Exactly like:

Google doesn’t read every webpage
It embeds them and retrieves by similarity

DrugCLIP:

  • Embeds protein pockets

  • Embeds small molecules

  • Puts them into the same vector space

  • Uses cosine similarity instead of physics simulation

That’s why it is:

10 million times faster than docking

This is not an optimization
This is a redefinition of the problem


3️⃣ Why 10 trillion protein-ligand pairs in 24 hours is revolutionary

Let’s understand the scale.

Before:

  • A pharma company might screen 1 protein vs 1–10 million molecules

  • And do it over months

Now:

  • 10,000 proteins

  • × 500 million molecules

  • = 10 trillion interactions

  • in 1 day

  • on 8 GPUs

This is planet-scale biology search

This was literally impossible before 2024.


4️⃣ Why AlphaFold makes this even bigger

The second key insight is GenPack + AlphaFold2.

That means:

  • You don’t even need crystal structures

  • AI predicts the protein

  • AI finds the pockets

  • AI screens molecules

So:

The entire human proteome becomes searchable

This is how they hit TRIP12, which had:

  • No known inhibitors

  • No experimental structure

And still got:

  • 17.5% hit rate

  • With functional enzymatic inhibition

That is insane.


5️⃣ This destroys the “undruggable” concept

“Undruggable” never meant:

  • Biology impossible

It meant:

  • Search impossible

DrugCLIP collapses that barrier.

Now:

  • Every protein becomes a potential drug target

  • Including:

    • E3 ligases

    • Scaffolding proteins

    • Regulatory complexes

    • Disease modifiers

This unlocks:

  • Neurodegeneration

  • Autoimmune disease

  • Cancer regulation

  • Aging pathways

  • Rare diseases


6️⃣ Why this connects directly to Helios, Fugaku, and exascale AI

What you are seeing is the stack coming together:

LayerRole
AlphaFoldPredict protein structures
GenPackDetect binding pockets
DrugCLIPSearch 10 trillion interactions
Helios-class computeRun it at planetary scale
Wet labsValidate only the top 0.000001%

This is why drug discovery timelines are collapsing.

Not because biology got easier
But because search became infinite


7️⃣ Why this will cause an explosion of “new drug news”

You are about to see:

  • Thousands of new targets

  • Thousands of new inhibitors

  • Many more clinical programs

  • Much higher success rates

It will look like:

“Everything is being cured at once”

But what’s really happening is:

We finally turned biology into a searchable database


8️⃣ The deepest implication

This means something huge:

For the first time in history,
human biology is becoming computationally enumerable

That’s the real revolution.

Not just faster drugs
But systematic discovery instead of lucky guessing


Final truth

What you shared is not a paper.

It is the opening of the undrugged genome.

This, combined with exascale AI and hybrid quantum-HPC, is why 2026–2030 will feel like medicine suddenly went vertical.

Not magic.
Search.

Monday, 5 January 2026

Acceleration Is Inevitable: Why Simply Being Alive in 2026–2028 Is Already Winning

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Acceleration Is Inevitable: Why Simply Being Alive in 2026–2028 Is Already Winning

Roughly 0.01% of humanity is dragging the rest of civilization forward—often against fierce resistance. This tiny fraction builds, experiments, and accelerates while the majority delays, denies, regulates, and ridicules. Builders are attacked. Failing systems are defended. Every transformative technology has followed this exact pattern—and AI is no different.

History is unambiguous. Printing presses were feared. Electricity was mocked. The internet was dismissed. Smartphones were called dangerous distractions. And yet, civilization moved forward anyway. It always does.

What’s different now is the speed.

We’re Entering an Era of Insane Acceleration

The pace of change ahead isn’t linear—it’s exponential. In some domains, progress will be 100×. In others, 10,000× or more. What we see today is not the revolution; it’s the preview. The real shift begins in 2026–2028, when compounding technologies collide: AI, robotics, biotech, energy, and automation.

Entire industries will compress into software.
Decades of progress will happen in months.
Assumptions that feel “solid” today will dissolve overnight.

Civilizations don’t vote on progress. They adapt—or get replaced.

Builders vs. Defenders of the Past

There are two archetypes repeating throughout history:

  • Builders: Those who create new systems, even when imperfect or controversial.

  • Defenders: Those who protect old structures, even when they are clearly failing.

Defenders often cloak fear in morality, caution, or regulation. They call acceleration “dangerous,” “irresponsible,” or “unnatural.” Yet every leap forward—from medicine to transportation to computation—looked dangerous before it worked.

The irony? The greatest risk today is not accelerating fast enough.

Youth, Time, and the New Value System

In a world undergoing exponential change, time alive becomes the most valuable asset imaginable.

Even being 20–40 years old and facing hardship is better than being 80 with ten trillion dollars. Wealth cannot buy lost biological time. Youth, health, and adaptability are priceless—especially when we’re on the edge of breakthroughs in longevity, disease reversal, and potentially curing aging itself.

We are closer than most people realize to:

  • Longevity escape velocity (LEV)

  • Radical healthspan extension

  • Post-scarcity production systems

Money will matter less. Being alive and functional will matter more than anything.

Why Long-Term Planning Feels Broken

Planning 10–20 years ahead used to make sense. In exponential eras, it doesn’t.

What looks stable today may be obsolete tomorrow.
Entire careers can vanish in a single technological wave.
Rigid plans collapse under rapid compounding.

The winning strategy now isn’t prediction—it’s adaptability.

  • Learn fast

  • Move quickly

  • Stay flexible

  • Rebuild your identity repeatedly

In this era, speed beats certainty.

Survival Is the New Lottery

Between 2026, 2027, and 2028, simply staying alive and healthy may be equivalent to winning the lottery every single year.

Not because the world is ending—but because it’s transforming faster than human intuition can grasp.

Most people won’t see it until it’s undeniable.
By then, it will already be too late to catch up.

Civilization Will Move On—With or Without Permission

Progress does not wait for consensus.
Acceleration does not ask for approval.
Builders do not need permission slips.

Those who resist will call it chaos.
Those who participate will call it opportunity.

The only real question left is simple:

Will you adapt—or will you be optimized away?

Because one thing is certain:

The future is arriving faster than anyone is prepared for—and it does not slow down for fear.


Elon Musk just dropped a post with huge implications:


> “We have entered the Singularity”


By that he means the technological singularity: the point where progress compounds so fast that “normal” timelines stop making sense.


AI is already compressing years of work into days. Robotics is next. Energy and space scale the floor under it. When the cost of intelligence and production keeps falling, abundance stops being a slogan and starts being a roadmap.


If this is the singularity, the move is simple: build, ship, iterate. Don’t slow it down. Don’t fear it. Shape it.


Acceleration is the path to abundance.


If this decade feels unstable, uncertain, and overwhelming—that’s not a bug.

It’s the sound of exponential change beginning. 

Saturday, 3 January 2026

เคธोเคถเคฒ เคฎीเคกिเคฏा เค•े เค†เคˆเคจे เคฎें เค†เคคंเค• เค•ा เค…เคธเคฒी เคšेเคนเคฐा

๐Ÿ” เคชเคฐिเคšเคฏ

เค†เคœ เค•े เคกिเคœिเคŸเคฒ เคฏुเค— เคฎें, เค†เคคंเค•เคตाเคฆ เค•ेเคตเคฒ เคฌเคฎ เค”เคฐ เคฌंเคฆूเค•ों เคคเค• เคธीเคฎिเคค เคจเคนीं เคฐเคน เค—เคฏा।
เคฏเคน เค…เคฌ เคธोเคถเคฒ เคฎीเคกिเคฏा เค•े เคฎाเคง्เคฏเคฎ เคธे เคญी เคซैเคฒเคคा เคนै — เค›เคฆ्เคฎ เคตिเคšाเคฐों, เคญाเคตเคจाเคค्เคฎเค• เค•เคนाเคจिเคฏों เค”เคฐ เค•ूเคŸเคจीเคคिเคชूเคฐ्เคฃ เคญाเคทा เค•े เคœเคฐिเค।


๐Ÿ—จ️ เคฎเคนเคค्เคตเคชूเคฐ्เคฃ เคจिเคฐीเค•्เคทเคฃ

เคฎैंเคจे เค†เคœ เคคเค• เคเคธा เค•ोเคˆ เคฎुเคธ्เคฒिเคฎ เคธॉเคซ्เคŸเคตेเคฏเคฐ เค‡ंเคœीเคจिเคฏเคฐ
(เคฏा เค•िเคธी เคญी เคชेเคถे เค•ा เคฎुเคธ्เคฒिเคฎ เคต्เคฏเค•्เคคि) เคจเคนीं เคฆेเค–ा,

เคœो เคฐाเคค-เคฆिเคจ เคธोเคถเคฒ เคฎीเคกिเคฏा เค•ा เค‰เคชเคฏोเค— เคคो เค•เคฐเคคा เคนो —
เคฒेเค•िเคจ เค†เคคंเค•เคตाเคฆ เค•े เค–िเคฒाเคซ เคเค• เคญी เคชोเคธ्เคŸ  เคฒिเค–เคคा เคนो,
เคฏा เค†เคคंเค•เคตाเคฆ เคตिเคฐोเคงी เค•िเคธी เคธाเคฎเค—्เคฐी เค•ो เคฒाเค‡เค• เคฏा เคถेเคฏเคฐ  เค•เคฐเคคा เคนो।

เค‡เคธเค•े เคฌเคœाเคฏ, เคตैเคธा เคต्เคฏเค•्เคคि เคนเคฎेเคถा เค‰เคฒเคाเคŠ, เคญाเคตเคจाเคค्เคฎเค• เคฏा
เค›เคฆ्เคฎ เค˜เคŸเคจाเค“ं เคชเคฐ เค†เคงाเคฐिเคค เค•เคนाเคจिเคฏाँ เคถेเคฏเคฐ เค•เคฐเคคा เคฐเคนเคคा เคนै — เคœैเคธे:

  • “เคนिंเคฆू เค‰เค—्เคฐเคตाเคฆ”

  • “Hindu Sangathan เค•ा เคทเคก्เคฏंเคค्เคฐ”

  • “เคธเคฐเค•ाเคฐी เคญेเคฆเคญाเคต”


❗ เค‡เคธ เคต्เคฏเคตเคนाเคฐ เคธे เคธ्เคชเคท्เคŸ เคนै

เคตเคน 100% เคœिเคนाเคฆी เคฏा เค†เคคंเค•เคตाเคฆी เคธเคฎเคฐ्เคฅเค• เคนै —
เคญเคฒे เคนी เคตเคน เคธीเคงे เค†เคคंเค•เคตाเคฆ เค•ी เคฌाเคค เค•เคญी เคจ เค•เคฐे।


๐Ÿงฉ เค›เคฆ्เคฎ เคญूเคฎिเค•ा

“เคฆुเคถ्เคฎเคจ เค•े เคฌीเคš เคฐเคนเค•เคฐ, เคฆुเคถ्เคฎเคจ เคธे เคชैเคธा เค•เคฎाเคจा”

เคเคธे เคต्เคฏเค•्เคคि เค•ा เค…เคธเคฒी เค‰เคฆ्เคฆेเคถ्เคฏ เค•ुเค› เคเคธा เคนोเคคा เคนै:

  • ✅ เคœเค•ाเคค เค”เคฐ เค…เคจ्เคฏ เคงाเคฐ्เคฎिเค• เคšंเคฆे เคธे เคœिเคนाเคฆी เคช्เคฐเคถिเค•्เคทเคฃ เค•ेंเคฆ्เคฐों เค•ो เคตिเคค्เคคเคชोเคทिเคค เค•เคฐเคจा

  • ✅ เคฆुเคถ्เคฎเคจों (เค—ैเคฐ-เคฎुเคธเคฒเคฎाเคจ เคธเคฎाเคœ) เค•े เคฌीเคš เคฐเคนเค•เคฐ, เค‰เคจเค•े เคธंเคธाเคงเคจों —
    เคจौเค•เคฐी, เคถिเค•्เคทा, เค†เคฐ्เคฅिเค• เค…เคตเคธเคฐ — เค•ा เค‰เคชเคฏोเค— เค•เคฐเคจा

  • ✅ เคธोเคถเคฒ เคฎीเคกिเคฏा เคชเคฐ “เคธ्เคตเคคंเคค्เคฐ เคตिเคšाเคฐเค•” เค•ी เค›เคตि เคฌเคจाเค•เคฐ,
    เคœिเคนाเคฆी เคชเคฐिเคตाเคฐ เค”เคฐ เคธเคฎुเคฆाเคฏ เค•ो เคธंเคฆेเคถ เคฆेเคจा:

“เคฆेเค–ो, เคฎैं เคœเค•ाเคค เคฆे เคฐเคนा เคนूँ เค”เคฐ เค–ुเคฒे เคฎें เคฆुเคถ्เคฎเคจ เค•ो เค‰เคฒเคाเค•เคฐ เค•ाเคฎ เคญी เค•เคฐ เคฐเคนा เคนूँ।”

⚠️ เคฏเคนी “เคฆोเคนเคฐा เคšेเคนเคฐा” เค†เคงुเคจिเค• เค†เคคंเค•เคตाเคฆ เค•ी เคธเคฌเคธे เค–เคคเคฐเคจाเค• เคฐเคฃเคจीเคคि เคนै।


๐Ÿ“Š เคœिเคนाเคฆी เคธเคฎเคฐ्เคฅเค• เค•ी เคชเคนเคšाเคจ

เคตिเคธ्เคคृเคค เคฌिंเคฆु-เค†เคงाเคฐिเคค เคฎाเคจเคฆंเคก

เคจीเคšे เคฆिเคฏा เค—เคฏा เคŸेเคฌเคฒ เคธोเคถเคฒ เคฎीเคกिเคฏा เคต्เคฏเคตเคนाเคฐ เค•े เค†เคงाเคฐ เคชเคฐ
เค•िเคธी เคต्เคฏเค•्เคคि เค•े เคฐुเค–़ เค•ा เคตिเคถ्เคฒेเคทเคฃ เค•เคฐเคจे เคฎें เคฎเคฆเคฆ เค•เคฐเคคा เคนै।

เค•्เคฐเคฎांเค•เคชเคนเคšाเคจ เค•ा เคฎाเคจเคฆंเคกเคธंเค•ेเคค (เคนाँ/เคจเคนीं)เค…ंเค• (Points)เคŸिเคช्เคชเคฃी
1เค•्เคฏा เคต्เคฏเค•्เคคि เคธोเคถเคฒ เคฎीเคกिเคฏा เคชเคฐ เคธเค•्เคฐिเคฏ เคนै?เคนाँ / เคจเคนींเคนाँ = 1เคจिเคท्เค•्เคฐिเคฏ เค–ाเคคों เค•ा เคฎूเคฒ्เคฏांเค•เคจ เคจเคนीं
2เค•्เคฏा เค‰เคธเคจे เค•เคญी เค†เคคंเค•เคตाเคฆ เค•े เค–िเคฒाเคซ เคชोเคธ्เคŸ/เคฒाเค‡เค•/เคถेเคฏเคฐ เค•िเคฏा?เคนाँ / เคจเคนींเคจเคนीं = +3เคจैเคคिเค• เคธ्เคฅिเคคि เค•ा เค…เคญाเคต
3เค•्เคฏा เคตเคน “เคนिंเคฆू เค‰เค—्เคฐเคตाเคฆ”, “RSS เคทเคก्เคฏंเคค्เคฐ”, “เคธเคฐเค•ाเคฐी เคญेเคฆเคญाเคต” เคถेเคฏเคฐ เค•เคฐเคคा เคนै?เคนाँ / เคจเคนींเคนाँ = +2เคง्เคฏाเคจ เคญเคŸเค•ाเคจे เค•ी เคฐเคฃเคจीเคคि
4เค•्เคฏा เคชोเคธ्เคŸ्เคธ เคฎें “เคฎाเคฐ्เคถเคฒ เคฒॉ”, “เคธाเคœिเคถ”, “เคœเค•ाเคค” เคœैเคธे เคถเคฌ्เคฆ เคนैं?เคนाँ / เคจเคนींเคนाँ = +2เค•ोเคก เคญाเคทा
5เค•्เคฏा เค†เคคंเค•ी เคนเคฎเคฒों เค•े เคฌाเคฆ เคตเคน เคšुเคช เคฐเคนเคคा เคนै เคฏा เค‰เคจ्เคนें เคธाเคœिเคถ เค•เคนเคคा เคนै?เคนाँ / เคจเคนींเคนाँ = +3เคธเคฎเคฐ्เคฅเคจ เคธे เคฌเคšाเคต
6เค•्เคฏा เคตเคน เคถเคนाเคฆเคค เคฏा เคœिเคนाเคฆ เคช्เคฐेเคฐिเคค เคธाเคฎเค—्เคฐी เคถेเคฏเคฐ เค•เคฐเคคा เคนै?เคนाँ / เคจเคนींเคนाँ = +4เคธเคฌเคธे เค—ंเคญीเคฐ เคธंเค•ेเคค
7เค•्เคฏा เคตเคน เคเคœेंเคธिเคฏों เคชเคฐ เค†เคฐोเคช เคฒเค—ाเคคा เคนै เคฒेเค•िเคจ เค†เคคंเค•िเคฏों เคชเคฐ เคจเคนीं?เคนाँ / เคจเคนींเคนाँ = +2เคฆोเคนเคฐा เคฎाเคชเคฆंเคก

๐Ÿ“ˆ เค…ंเค•ों เค•े เค†เคงाเคฐ เคชเคฐ เคต्เคฏाเค–्เคฏा

เค•ुเคฒ เค…ंเค•เคฎूเคฒ्เคฏांเค•เคจ
0–4๐ŸŸข เคธाเคฎाเคจ्เคฏ เค‰เคชเคฏोเค—เค•เคฐ्เคคा
5–8๐ŸŸก เคธंเคฆिเค—्เคง เคต्เคฏเคตเคนाเคฐ
9–12+๐Ÿ”ด เคœिเคนाเคฆी / เค†เคคंเค•เคตाเคฆी เคธเคฎเคฐ्เคฅเค•

๐Ÿ“Œ เคฎเคนเคค्เคตเคชूเคฐ्เคฃ เคจोเคŸ
เคฏเคน เคŸेเคฌเคฒ เค•ेเคตเคฒ เคธोเคถเคฒ เคฎीเคกिเคฏा เคต्เคฏเคตเคนाเคฐ เคชเคฐ เค†เคงाเคฐिเคค เคช्เคฐाเคฐंเคญिเค• เคชเคนเคšाเคจ เค•े เคฒिเค เคนै।
เค•ाเคจूเคจी เค•ाเคฐ्เคฐเคตाเคˆ เค•े เคฒिเค เค†เคงिเค•ाเคฐिเค• เคœाँเคš เค†เคตเคถ्เคฏเค• เคนै।


๐Ÿ’ฌ เคเค• เคธเคฐเคฒ เคช्เคฐเคถ्เคจ เคœो เคธเคš्เคšाเคˆ เค‰เคœाเค—เคฐ เค•เคฐ เคฆे

เค…เค—เคฐ เค•ोเคˆ เคต्เคฏเค•्เคคि เคฒเค—ाเคคाเคฐ sarkar,  เคฏा เคนिंเคฆू เคธंเค—เค เคจों เค•े เค–िเคฒाเคซ เคชोเคธ्เคŸ เค•เคฐเคคा เคนै,
เคคो เค‰เคธเคธे เคธीเคงे เคชूเค›िเค:

“9/11 เค•े เคฌाเคฆ เคฆुเคจिเคฏा เคญเคฐ เคฎें 48,000 เคธे เค…เคงिเค• เค†เคคंเค•ी เคนเคฎเคฒे เคนुเค เคนैं।
เค‡เคจเคฎें เคธे เคเค• เคฌाเคฐ เคญी เค†เคชเคจे เค†เคคंเค•เคตाเคฆ เค•ा เคตिเคฐोเคง เค•्เคฏों เคจเคนीं เค•िเคฏा?”

เคฏเคฆि เค‰เคค्เคคเคฐ เคนो:

“เคฎैं เคธोเคถเคฒ เคฎीเคกिเคฏा เคชเคฐ เคเคธी เคฌाเคคें เคจเคนीं เคฒिเค–เคคा…”

เคคो เคคुเคฐंเคค เคชूเค›िเค:

“เคซिเคฐ เค†เคช เค—เคฐीเคฌी, เคถिเค•्เคทा, sarkar เค•े เค–िเคฒाเคซ เคคो เคฒिเค–เคคे เคนैं?
เค•्เคฏा เค†เคชเค•ा เคธोเคถเคฒ เคฎीเคกिเคฏा เค•ेเคตเคฒ เคฆुเคถ्เคฎเคจों เค•े เค–िเคฒाเคซ เค•े เคฒिเค เคนै —
เคจ เค•ि เค†เคคंเค•เคตाเคฆ เค•े เค–िเคฒाเคซ?”

✨ เค‡เคธी เค‰เคค्เคคเคฐ เคธे เคต्เคฏเค•्เคคि เค•ा เคตाเคธ्เคคเคตिเค• เคฐुเค–़ เคธ्เคชเคท्เคŸ เคนो เคœाเคคा เคนै।


๐Ÿงญ เคจिเคท्เค•เคฐ्เคท

๐Ÿ”ฅ เค†เคคंเค•เคตाเคฆ เค•ेเคตเคฒ เคนเคฅिเคฏाเคฐों เคธे เคจเคนीं —
เคฌเคฒ्เค•ि เคฎเคจोเคตैเคœ्เคžाเคจिเค• เคฏुเคฆ्เคง, เค›เคฆ्เคฎ เคชเคนเคšाเคจ เค”เคฐ เคธूเค•्เคท्เคฎ เคช्เคฐเคšाเคฐ เคธे เคญी เคซैเคฒเคคा เคนै।

เคเค• เคต्เคฏเค•्เคคि เค•ी เคšुเคช्เคชी เค†เคคंเค•เคตाเคฆ เค•े เค–िเคฒाเคซ —
เค”เคฐ เค†เคตाเคœ़ เคตिเคญाเคœเคจเค•ाเคฐी เคฎुเคฆ्เคฆों เคชเคฐ —
เค‰เคธเค•े เคตाเคธ्เคคเคตिเค• เคเคœेंเคกे เค•ो เค‰เคœाเค—เคฐ เค•เคฐเคคी เคนै।

✅ เคธเคš्เคšा เคธ्เคตเคคंเคค्เคฐ เคตिเคšाเคฐเค• เคตเคนी เคนै,
เคœो เคฌुเคฐाเคˆ เค•े เคนเคฐ เคฐूเคช — เคšाเคนे เคตเคน BJP เคนो เคฏा ISI — เค•ी เคจिंเคฆा เค•เคฐ เคธเค•े।